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following exposure, micedevelop many of the same pathological signs, includingloss of the neurotransmitter dopamine and dopamineneurons in the substantia nigra pars compacta as well asbehavioral symptoms, such as reduced locomotion that areroutinely observed in patients with PD.

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These are weak points where vasa recta passthrough the muscularis propria and penetrate the submucosa. As an exception to the rule of PMN pleocyto-sis, Streptococcus spp. Reflection and evidence-based practicein action: A case based application. Still later (4–8 years) the ‘on-off’ phenomenaand marked dyskinesias may become so prominentthat the patient is as incapacitated with the drugas without it. These same authors later demonstrated thateven relatively young (20–39 years) ?4 homozygotes hadabnormally low rates of glucose metabolism bilaterally inthe posterior cingulate, parietal, temporal, and prefron-tal cortex, and that the ?4-gene dose is correlated withlower glucose metabolism in each of these brain regions(see Figure 7.9; Reiman et al., 2004, 2005)

These same authors later demonstrated thateven relatively young (20–39 years) ?4 homozygotes hadabnormally low rates of glucose metabolism bilaterally inthe posterior cingulate, parietal, temporal, and prefron-tal cortex, and that the ?4-gene dose is correlated withlower glucose metabolism in each of these brain regions(see Figure 7.9; Reiman et al., 2004, 2005). In order to call (or‘upgrade’) a potential risk factor a causal factor, several pieces of evidence arerequired

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